Aurelia Crețu Elena Cristina Mitrofan

Abstract

The pulmonary cavity may reflect the presence of a broad spectrum of lung pathological processes, with surprising etiology at times. Bacteria, fungi, viruses, parasites, embolism, malignancy, and autoimmune disease may be involved in its formation. We present the case of a 78 year-old patient who was hospitalized 7 days prior to admission to our hospital with grade IV mMRC dyspnea and dry cough, the chest CT indicating pulmonary embolism and the presence of a pulmonary cavity, suggestive of neoplastic etiology. An infection with Staphylococcus aureus complicated with pulmonary embolism, two pathological entities which can lead to cavitary pulmonary damage, can prove challenging to any physician in the given context.

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Keywords

lung cavity, Staphylococcus aureus, pulmonary embolism

References
1. Gadkowski LB, Stout JE. Cavitary pulmonary disease. Clin Microbiol Rev 2008; 21(2):305–333.
2. Gafoor K, Patel S, Girvin F, et al. Cavitary lung diseases – A clinical-radiological algorithmic approach. Chest 2018; 153(6):1443-1465.
3. Gerada E, Gatt N, Mizzi A, Montefort S. Atypical cavitary lung lesions: a case report and review of radiologic manifestations. Intern Med 2014; 4:131.
4. Gill RR, Matsusoka S, Hatabu H. Cavities in the lung in oncology patients: imaging overview and differential diagnoses. Appl Radiol 2010; 39(6):10-21.
5. Mayberry JP, Primack SL, Müller NL. Thoracic manifestations of systemic autoimmune diseases: radiographic and high-resolution CT findings. Radio Graphics 2000; 20(6):1623-1635.
6. Serrano Usaola N, Martin Egaña M, Beltran de Otalora Garcia S, et al. Cavitated lung lesions. A diagnostic approach. European Society of Radiology ECR 2013/C-0351 2013; 1-24. Doi: 10.1594/ecr2013/C-0351.
7. Brown ML, O'Hara FP, Close NM, et al. Prevalence and sequence variation of panton-valentine leukocidin in methicillin-resistant and methicillin-susceptible Staphylococcus aureus strains in the United States. J Clin. Microbiol 2012; 50(1):86-90.
8. Chatha N, Fortin D, Bosma KJ. Management of necrotizing pneumonia and pulmonary gangrene: A case series and review of the literature. Can Respir J 2014; 21(4):239–245.
9. Chou DW, Wu SL, Chung KM, Han SC, Cheung BM. Septic pulmonary embolism requiring critical care: clinicoradiological spectrum, causative pathogens and outcomes. Clinics 2016; 71(10):562–569.
10. Manafi A, Khodabandehloo M, Rouhi S, et al. Molecular epidemiology survey of Staphylococcus aureus panton-valentine leukocidin-positive isolated from Sanandaj, Iran. Adv Biomed Res 2017; 6:87.
11. Ieven M, Coenen S, Loens K, et al. Aetiology of lower respiratory tract infection in adults in primary care: a prospective study in 11 European countries. Clin Microbiol Infect 2018; 1.e1-1.e6.
12. Kumar DR, Hanlin E, Glurich I, Mazza JJ, Yale SH. Virchows contribution to the understanding of thrombosis and cellular biology. Clin Med Res 2010; 8(3-4):168–172.
13. Méan M, Limacher A, Stalder O, et al. Do Factor V Leiden and Prothrombin G20210A mutations predict recurrent venous thromboembolism in older patients? Am J Med 2017; 130(10):1220.e17-1220.e22.
How to Cite
Crețu, A., & Mitrofan, E. C. (2018). Approaching cavitary lung lesion in a patient with pulmonary embolism. Archive of Clinical Cases, 5(4), Arch Clin Cases 2018; 5(4):165-171. https://doi.org/10.22551/2018.21.0504.10145
Section
Case Reports

How to Cite

Crețu, A., & Mitrofan, E. C. (2018). Approaching cavitary lung lesion in a patient with pulmonary embolism. Archive of Clinical Cases, 5(4), Arch Clin Cases 2018; 5(4):165-171. https://doi.org/10.22551/2018.21.0504.10145